Let’s look at what diabetes is first.
Chronic hyperglycemia, which persists during diabetes, leads to chronic damage and dysfunction of various tissues, especially the eyes, kidneys, heart, blood vessels and nerves.
Among the foods that people eat into it are starchy foods, what we call staple foods.
The food made its way into the stomach for a short time and into the small intestine. The small intestine breaks down starch into glucose through the action of digestive enzymes. Glucose enters the bloodstream and becomes blood sugar.
The blood is like a little train, carrying sugar down the street and into every cell. Glucose is an important source of energy for cells and most of the energy needed for all cellular activities comes from sugar.
The cell is also very good at getting by, it consumes a portion of the sugar that supplies daily life and stores the portion that it can’t eat in the form of fat to eat when it’s hungry.
That’s pretty much what sugar does in the body.
Is it that simple? Of course not, and how many other important people didn’t make an appearance?
The first thing to understand is the cell, the cell is very delicate, not sugar can get in just because it wants to, only by opening the door of the cell can sugar get in, and the key to open this door is insulin.
Insulin is the only hormone that lowers blood sugar.
If the sugar can’t get into the cells it stays in the blood and the blood is overloaded with sugar.
The pancreas is an important producer of insulin, so how does it know when it needs to secrete insulin?
The other key player, that’s pancreatic hypertriglyceride-like peptide I. Let’s call him “Peptune”.
Peptide Jun is produced at the same time as the small intestine breaks down starch, and it has one function, to report the news.
Peptide Jun will be the first to report to the pancreas the news that blood sugar is going to rise: “You hold on tight and hurry up your insulin secretion.”
At this point, the pancreas begins to secrete insulin.
The life cycle of insulin is only 5-15 minutes and its only mission is to find the cells to open the door for sugar to enter the cells and supply energy to the cells so that when the sugar has a way out, the sugar content in the blood will be lower.
So what’s the deal with diabetes?
Some people have an absolute lack of insulin secretion due to factors like immune system defects and heredity, then sugar can’t get into the cells smoothly and the blood sugar stays over the limit.
Type 1 diabetes accounts for a relatively small percentage, about 5-10% of all diabetics, and it has a distinctive feature – early onset, usually in childhood or adolescence.
In addition to type 1 diabetes, there is also type 2 diabetes, which is the most prevalent, most complex, hidden and deeper in the diabetic population, and type 2 diabetes is the main form of diabetes, accounting for about 90% of the total.
So what’s the deal with type 2 diabetes?
In the process of sugar metabolism, when the small intestine is breaking down starch, if it forgets to secrete peptide kun, that is, there is no communicator to report the news, the pancreas will sleep at home foolishly, not knowing to secrete insulin.
This type of hyperglycemia, caused by an error in communication facilities, is a type 2 diabetes.
Of course, this is a very absolute situation, more often than not, the small intestine secretes peptide as usual, and the peptide does not work actively, slow to report, the pancreas is also slow to secrete after receiving orders, and the secreted insulin is slow to open the door again.
If one of the three guys, Peptide Jun, Pancreas, and Insulin, suffer from procrastination, or all of them, it’s a bad outcome.
When blood glucose rises rapidly, insulin does not come; when sugar is excreted with urine and blood glucose decreases in the body, these insulins come late and start to work hard, so that blood glucose, which is already not high, drops instantly.
Healthy people use insulin to regulate their blood sugar levels to keep them as relatively stable a state as possible.
That’s great, because of their procrastination, our blood sugar has been on a roller coaster, either super high or super low.
The most important thing is that you can’t afford to have a high blood sugar fluctuation, and a low blood sugar can make you faint immediately, or even …….
If in the process of sugar metabolism, peptide jun painstakingly ventilates the pancreas on time, the pancreas receives the notice and begins to secrete insulin.
Suddenly it turns out, oops my god, productivity is limited and the quantity is not up to par, this time some of the sugar does not enter the cells smoothly and stays in the blood, causing a high blood sugar condition, which is also a type 2 diabetes.
If, during the process of sugar metabolism, the intelligence work of peptide and the production of the pancreas are smooth, but I never thought that the ability of the insulin to open the door was missing.
That is, you are sent an electronic access card, someone else’s card is swiped, the door is opened, but your card just does not work.
Because the insulin receptors are not sensitive enough, one cannot be brushed, two cannot be brushed…… The tragedy is that if you can’t open the door, your blood sugar will go up!
There is also a condition that the digestive function of the gastrointestinal is really too good to eat the starch seconds into the blood sugar.
The instant spike in blood sugar makes insulin invulnerable, causing a large amount of glucose to build up in the blood, causing high blood sugar.
So, the pancreas, insulin, peptide, stomach and intestines are the main characters that either malfunctioned themselves or malfunctioned in the pairing, which led to the high blood sugar and diabetes.
Content reference deep reading video articles.